Each morphology asks for its own protocol.
The way certain spots come back. The product cycle that fixed one thing and made another worse. The clinic visit where “acne” was the word you were given and the regimen didn’t quite fit. Most patients arrive here with one word for what they’re seeing in the mirror. The skin tells a more specific story.
Comedonal
Blackheads (open comedones) and whiteheads (closed comedones). The clogged-pore mechanism without the inflammatory cascade. Often the earliest pattern, and often what is left to clear once inflammatory acne has quieted. Comedonal acne responds to surface refinement and to topical-procedural sequencing.
Inflammatory papules and pustules
The red and yellow bumps. C. acnes proliferation in the follicular unit plus inflammation in the surrounding tissue. The pattern most patients are searching when they search “acne”. Heightened by hormones, friction, occlusive products, and humidity. Lesions resolve and recur in cycles until the inflammatory drive is brought under control.
Hormonal-driven
The lower-face, jawline, and neck pattern that follows monthly cycles or adult-onset hormonal shifts. May trace to idiopathic adult female hormonal acne, perimenopausal shifts, or PMOS (Polyendocrine Metabolic Ovarian Syndrome, formerly PCOS). The cause sits with a GP or endocrinologist; the inflammatory presentation and the PIH that follows it is what Pink treats. We work alongside the prescribing pathway, not around it.
Acne mechanica
Acne that follows friction or pressure. Masks, helmets, shoulder bag straps, phone-on-jawline habits, gym equipment contact zones. Distinguished by where it lands rather than by lesion morphology. Resolution starts with removing the stimulus. Once the cause is identified, we shorten the timeline with PinkRX peels, MediSOL LED, and Nd:YAG where the inflammatory burden warrants it.
Post-inflammatory hyperpigmentation (PIH)
The brown, tan, or grey-brown flat marks left where breakouts used to be. Melanin, not architecture. Not a scar. Common on skin types IV through VI where baseline melanocyte activity amplifies any inflammatory signal. The Australasian College of Dermatologists is explicit that this is not scarring. PIH responds to peel sequencing and laser support.
Post-inflammatory erythema (PIE)
The pink-to-red flat marks left where breakouts used to be. Vascular, not architecture. Not a scar. More visible on lighter skin where vascular events read through. Often fades on its own across three to twenty-four months. Where it persists past six months it usually needs targeted treatment.
Most patients have a mix. The plan is matched to the mix.
The acne-type guide that follows names what we treat and how. Where a pattern belongs with a dermatologist instead of with us, we say so plainly in honest scoping below.